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Additionally, hyaluronic acid (HA)-modified cell membranes have been reported to specifically target M1 macrophages that overexpress CD44 receptors, thereby enhancing the efficacy of targeted therapy [32]

GLP-1(7-36) inhibits atherosclerosis progression in ApoE-/- mouse aorta by lowering expressions of CD36 and ACAT1 in foam cells

Caradonna, L., Amati, L., Magrone, T., Pellegrino, N., Jirillo, E., & Caccavo, D

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Given that the IGF pathway plays an important role in the development and progression of CRC, genetic changes affecting pathway members may play a role in the risk to develop CRC

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